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Monitoring respiratory function after administering opioids or ketamine is critical because both of these medications can cause respiratory depression, potentially leading to inadequate ventilation and oxygenation. Opioids, in particular, are known for their ability to depress the central nervous system, which affects breathing patterns. Ketamine, while sometimes used for its dissociative anesthetic properties, can also influence respiratory function, particularly at higher doses or in certain patient populations.

Immediate monitoring of respiratory rate, depth of respiration, and overall airway patency is essential to ensure that the patient maintains adequate respiratory function. If signs of respiratory distress or failure are observed, timely interventions such as airway management or supplemental oxygen may be required to prevent complications.

Other options, while relevant to patient care, do not carry the same immediate concern in the context of opioid or ketamine administration. Visual acuity, gastrointestinal function, and temperature regulation are important in a broader context but are not the primary focus of monitoring following the use of these sedatives and analgesics.